Summary: Background: SOD1 mutations are a significant contributor of familial amyotrophic lateral sclerosis (ALS) cases. SOD1 mutations increase the propensity for the protein to misfold and aggregate into insoluble proteinaceous deposits within motor neurons and neighbouring cells. The small molecule. CuATSM. https://bkirixgdxkd69.timeblog.net/70682926/utilizing-social-media-to-study-information-seeking-and-ethical-issues-in-gene-therapy